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Oxidative distress causes damage to fundamental macromolecules including proteins, lipids, RNA and DNA at a cellular level, which promotes abnormal cell death and inflammation, often culminating in additional oxidative stress at a systemic level

In diabetic rats, which are known to exhibit severely delayed wound healing, PIC treatment demonstrated elevated glutathione and ascorbic acid levels, improved epithelialization, and enhanced activation of fibroblasts and mast cells in the wound area

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Following, collected cells were dissolved in 200 L of Protein Extraction Reagent (ThermoFisher Scientific, Waltham, MA, USA) to extract the total cellular protein content